[8] | F. J. Cubero, G. Zhao, Y. A. Nevzorova, M. Hatting, M. Al Masaoudi, J. Verdier, J. Peng, F. M. Schaefer, N. Hermanns, M. V. Boekschoten, C. Grouls, N. Gassler, F. Kiessling, M. Muller, R. J. Davis, C. Liedtke, C. Trautwein, "Haematopoietic cell-derived Jnk1 is crucial for chronic inflammation and carcinogenesis in an experimental model of liver injury", J. Hepatol., vol. 62, no. 1, pp. 140–149.
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[7] | M. Hatting, M. Spannbauer, J. Peng, M. Al Masaoudi, G. Sellge, Y. A. Nevzorova, N. Gassler, C. Liedtke, F. J. Cubero, C. Trautwein, "Lack of gp130 expression in hepatocytes attenuates tumor progression in the DEN model", Cell Death Dis., vol. 6, no. 3, pp. e1667–e1667.
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[6] | M. Hatting, G. Zhao, F. Schumacher, G. Sellge, M. Al Masaoudi, N. Gaβler, M. Boekschoten, M. Müller, C. Liedtke, F. J. Cubero, C. Trautwein, "Hepatocyte caspase-8 is an essential modulator of steatohepatitis in rodents", Hepatology, vol. 57, no. 6, pp. 2189–2201.
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[5] | M. Hatting, A. K. Rines, C. Luo, M. Tabata, K. Sharabi, J. A. Hall, F. Verdeguer, C. Trautwein, P. Puigserver, "Adipose Tissue CLK2 Promotes Energy Expenditure during High-Fat Diet Intermittent Fasting", Cell Metab., vol. 25, no. 2, pp. 428–437.
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[4] | D. C. Kroy, F. Schumacher, P. Ramadori, M. Hatting, I. Bergheim, N. Gassler, M. V. Boekschoten, M. Müller, K. L. Streetz, C. Trautwein, "Hepatocyte specific deletion of c-Met leads to the development of severe non-alcoholic steatohepatitis in mice", J. Hepatol., vol. 61, no. 4, pp. 883–890.
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[3] | C. Liedtke, J. Bangen, J. Freimuth, N. Beraza, D. Lambertz, F. J. Cubero, M. Hatting, K. R. Karlmark, K. L. Streetz, G. A. Krombach, F. Tacke, N. Gassler, D. Riethmacher, C. Trautwein, "Loss of Caspase-8 Protects Mice Against Inflammation-Related Hepatocarcinogenesis but Induces Non-Apoptotic Liver Injury", Gastroenterology, vol. 141, no. 6, pp. 2176–2187.
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[2] | C. Luo, E. Balsa, A. Thomas, M. Hatting, M. Jedrychowski, S. P. Gygi, H. R. Widlund, P. Puigserver, "ERRα Maintains Mitochondrial Oxidative Metabolism and Constitutes an Actionable Target in PGC1α-Elevated Melanomas", Mol. Cancer Res., vol. 15, no. 10, pp. 1366–1375.
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[1] | F. Verdeguer, M. S. Soustek, M. Hatting, S. M. Blättler, D. McDonald, J. J. Barrow, P. Puigserver, "Brown Adipose YY1 Deficiency Activates Expression of Secreted Proteins Linked to Energy Expenditure and Prevents Diet-Induced Obesity", Mol. Cell. Biol., vol. 36, no. 1, pp. 184–196.
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